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Thus, the pathogenesis of gout is fundamentally driven by hyperuricemia, which serves as both a precursor and a central pathological hub

Senger B, Memar SA, Ahmann A, Houser JJ, Doughty-McDonald L

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G6PD activity was increased in postinfarct surviving myocardium and in failing myocardium in both human and animal models of disease (9, 32, 35, 38, 39, 45)

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