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Here, using an animal model for riboflavin deficiency (ariboflavinosis), we show that patients suffering from ariboflavinosis due to mutations in the riboflavin transporters have retinal changes that may not be reversible if they are not identified early and provided with riboflavin supplementation

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Salmonella pathogenicity island 2-encoded type III secretion system mediates exclusion of NADPH oxidase assembly from the phagosomal membrane

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The difference in OS between MCL patients carrying the TP53 mutation and patients without the mutation was considerable (17.6 months vs

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