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Overexpression of PHB2 can activate mitophagy, restore mitochondrial activity, enhance myocardial cell viability, and inhibit endotoxin-induced mitochondrial dysfunction in myocardial cells [15]

10.1007/s00011-023-01800-5 128 ZhangD.LiuB.CaoB.WeiF.YuX.LiG

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Once the neoplasms had developed, the mice were treated with low dose vinblastine and the antibody DC101, against the anti-murine VEGFR2 , or with the DC101 antibody alone