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Molecular formula: C78H123N23O23S2
The development and maintenance of sepsis-induced anomalies in cellular energy metabolism can be attributed to the decline in the expression of genes that encode crucial electron transport and glycolytic proteins ( in vitro studies ( The involvement of mitochondrial OS in the development of organ dysfunction induced by sepsis has been established
Horowitz MB, Purdy PD
Wang, F., Chen, F., Cai, Y., Zhang, G
