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It acts as a physiological ligand for Fe 2 + , promoting assembly of iron-sulfur (Fe-S) clusters and biosynthesis of Fe-S proteins, which participate in essential biological functions including electron transfer, the Krebs cycle, maintenance of genome integrity, enzymatic catalysis, and iron homeostasis 15
As NAD+ levels drop with age, so does mitochondrial function, DNA repair, and your bodys ability to manage inflammation and oxidative stress
doi:10.2478/fhort-2024-0002
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These sources contribute to the overall cellular redox balance, and their dysregulation can lead to oxidative stress and cellular damage
