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10.1038/s41392-025-02198-8 187 TremoliE.GhiselliG.MadernaP.ColliS.SirtoriC
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Using human brain organoid models derived from induced pluripotent stem cells (iPSCs) with targeted knockout of the CNTNAP2 gene, which is associated with autism spectrum disorder (ASD), it was revealed that CNTNAP2-deficient brain organoids exhibit abnormalities such as accelerated cell cycle, increased cortical folding, disrupted glutamatergic/GABAergic synaptic pathways, and upregulation of AKT/mTOR signaling (106)
ABC transporters in multidrug resistance and pharmacokinetics, and strategies for drug development
To view a copy of this licence, visit About this article Cite this article Mu, S., Lin, C., Skrzypczyk-Ostaszewicz, A
4a), indicating the enhanced flux of metabolites in the transsulfuration pathway
