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Mesothelioma tumor cells can be characterized as redox-driven tumors given the elevated levels of cellular ROS and increased expression of genes associated with escape from oxidative stress 7,9,10,11,12,13

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Enteroviral 2B protein has been shown to interact with mitochondrial voltage-dependent anion channel 3 (VDAC3) to induce ROS production, and knockdown of VDAC3 significantly reduces EV71-induced mtROS generation and viral replication 132