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These findings are consistent with those for other antioxidant enzymes: for example, lack of peroxiredoxin 3, a mitochondrially localized antioxidant enzyme, also augments apoptotic signaling in some cell types (60)

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against extrinsic apoptosis, necroptosis, and ferroptosis

B., Alexander J., Larsson A., Aaseth J

PPAR co-activator-1 (PGC-1) reduces amyloid- generation through a PPAR-dependent mechanism