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[Google Scholar] 152.Fernandes E.J., Poetini M.R., Barrientos M.S., Bortolotto V.C., Araujo S.M., Santos Musachio E.A., et al

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This hypothesis posits that with steatosis, the liver undergoes ER stress, oxidative stress and mitochondrial dysfunction, leading to an increase in oxidative metabolites, hepatocellular damage and secretion of a large number of inflammatory factors, which ultimately leads to further deterioration into liver disease, liver fibrosis, cirrhosis, inflammatory necrosis and other reactions (19,20)

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The oxidation steps, utilizing glucose-6-phosphate (G6P) as the substrate, occur at the beginning of the pathway and are the reactions that generate NADPH