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For in vivo assessment, MSCs with different cellular GSH levels or cell culture densities were injected in a rabbit chondral defect model, followed by histological analysis of cartilage-regenerated defect sites

Yang Y, Wang P, Guo J, Ma T, Hu Y, Huang L et al (2023) Zinc overload induces damage to H9c2 cardiomyocyte through mitochondrial dysfunction and ROS-mediated mitophagy

doi: 10.1016/S0098-2997(97)00008-3

Similarly, other studies suggest that inhibition of mitochondrial complex I activity and facilitation of -synuclein aggregation may be closely associated with rotenones selective dopaminergic toxicity in neurons [14,65]

Growth differentiation factor 15 in erythroid health and disease