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Methods Phase 1 involved 65 children who received one round of DMSA (3 days)

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A unifying genetic model for facioscapulohumeral muscular dystrophy

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78 AKT activation regulates cell survival, growth, and metabolism by inhibiting p27, activating cAMP-response element binding protein (CREB), inactivating tuberous sclerosis complex 2 (TSC2), sequestering FOXO in the cytoplasm, and engaging a feedback loop with mTORC2 (AKT and mTORC2 form a critical feedback loop in which mTORC2 activates AKT by phosphorylating it at serine (Ser)473

Dapsone- and nitroso dapsone-specific activation of T cells from hypersensitive patients expressing the risk allele HLA-B*13:01