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Molecular signaling and dysfunction of the human reactive enteric glial cell phenotype: implications for GI infection, IBD, POI, neurological, motility, and GI disorders

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doi: 10.1111/dme.14458 Ric Fordham, Ketan Dhatariya, Rachel Stancliffe, Adam Lloyd, Mou Chatterjee, Mevin Mathew, Loveleen Taneja, Mike Gains, Ulrik Haagen Panton

Thats where NAD+ complements the physiology of GLP-1/GIP therapy

It helps transport fatty acids into the mitochondria and provides a propionyl group involved in energy-generating pathways

This paper presents an interesting study describing defective hepatic thyroid hormone signalling in human NAFLD