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Daley AJ, Copeland RJ, Wright NP, Roalfe A, Wales JKH
The collapse of m not only reflects impaired electron transport and ATP synthesis but also serves as a molecular trigger for mitochondrial dysfunction and downstream apoptotic signaling (Liu et al., 2020)
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However, its research is limited, and it can be dangerous
Understanding the role of TDP-43 and FUS/TLS in ALS and beyond
The accumulated data indicate that mitochondria, mTOR, and autophagy may represent an integral regulatory axis during ASD [16, 251, 253, 308]
