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Finally, redox-based chemical readouts, such as for GR activity in the cerebrospinal fluid, could serve as a possible biomarker for PRKN mutation-linked disease, of its progression, and for the response to future interventions [31, 56, 58]

At the end of the treatment period, participants showed significant improvements in total NTSS6 scores with a particular reduction in deep aching and burning pain

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As they also demonstrated that PKC mediated morphine-induced MOR desensitization, it can be inferred that PKC would phosphorylate MOR at other sites than S363, T370, and S375