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The therapeutic efficacy of BM-MSCs can be significantly reduced by blocking the vagus nerve, administering drug inhibitors, or implementing gene knockout to inhibit CAP, and BM-MSC-derived prostaglandin E2 (PGE2) can promote acetylcholine (Ach) synthesis and release
there were no differences in ALT non-responders
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Together, they help convert destructive molecules, including free radicals, into harmless compounds
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It would remain the responsibility of Primary Care to diagnose and treat B12 deficient patients but they would have to choose to
