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The tripeptide maintains cellular redox homeostasis through the glutathione redox cycle, where NADPH-dependent glutathione reductase continuously regenerates GSH from GSSG

NO and GSNOR inhibitor increased the expression levels of PR1 , NPR1 , and TGA1 genes (Figure 6)

Cells 11 , 1897 (2022)

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Dart RC, Bailey E (2007)
