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doi: 10.1093/jas/skad354

found that HAC MSCs could inhibit the activation of inositol requiring enzyme 1 by reducing the expression of glucose regulated protein 78, effectively reversing endoplasmic reticulum stress (ER) in testicular tissue, reducing the production of apoptosis factors (e.g., Capase12 and Capase3) in rat seminiferous tubules caused by ER stress, eventually exerting an effect on mitigating radiation-induced testicular tissue damage in rat

10.3390/biom12081035 Summary Keywords alcohol-related liver disease, cell death patterns, intestinal flora, immune response, molecular mechanisms, therapeutic strategy, animal models Citation Hong X, Huang S, Jiang H, Ma Q, Qiu J, Luo Q, Cao C, Xu Y, Chen F, Chen Y, Sun C, Fu H, Liu Y, Li C, Chen F and Qiu P (2024) Alcohol-related liver disease (ALD): current perspectives on pathogenesis, therapeutic strategies, and animal models

"Glutathione Depletion Methyleation Cycle Block Hypothesis for the Pathogenesis of Chronic Fatigue Syndrome"

Moore T, Le A, Niemi AK, et al