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[PubMed] [Google Scholar] 247.Brosnahan SB, Jonkman AH, Kugler MC, Munger JS, Kaufman DA

Mechanisms by which metabolites of DCVC and other nephrotoxic haloalkene cysteine S -conjugates alter thiol status may involve direct inhibition of glutathione reductase (Lock and Schnellmann, 1990) and generation of oxidative stress that leads indirectly to oxidation of thiol compounds (Chen et al., 1990)

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Factors responsible for the induction of ferroptosis may directly or indirectly affect glutathione peroxidases