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In T2DM, elevated LC3-II and p62 levels suggest that autophagic flux is active but may be dysfunctional due to inefficient degradation of the autophagosomes
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Therefore, targeted blockade of lipophagy of HSCs should be an expected antifibrosis strategy
Hepatic GSH protein level was downregulated significantly after LPS induction, while BCSO was able to restore its level to normal
[DOI] [PubMed] [Google Scholar] 59.Zhu J, Li Y, Shen W, et al
Kelishadi R, Sabri M, Motamedi N, et al
