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The overproduction of inflammatory cytokines and the continuous recruitment of immune cells, coupled with the tissue damage caused by bacterial toxins, prevent effective immune clearance of the pathogens, impair tissue repair processes, and delay the healing of the wound

In addition to the above, cooperation of the HGF/c-Met pathway with MUCI (Tao et al., 2016) can induce hepatocarcinogenesis

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Figure 4 Histological examination of lung tissue from IL-33 challenged Rag2 - / - GC - / - mice revealed that adoptive transfer of activated ILC2s induced marked immune cell infiltration into the lung parenchyma and significant thickening of the airway epithelium compared with no-transfer controls ( Figures 4B, C, E, F )