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Insulin promotes macrophage phenotype transition through PI3K/Akt and PPAR-gamma signaling during diabetic wound healing

Death was verified by absence of spontaneous breathing, heartbeat, and corneal reflex

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The treatment control (RB) group, treated with Rhodamine B alone, exhibited minimal fluorescence intensity

The identified glutathione-related pathways provide potential therapeutic targets for alleviating oxidative stress-induced hippocampal injury in SAE, offering new insights for the clinical management of SAE-related neurological damage

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