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Figure 2 In the methylation cycle, methionine is activated by the addition of an adenosyl group from ATP to form S-adenosylmethionine (SAM)

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Because the inability to silence TEs results in genomic instability in germ cells, deficiencies in the piRNA pathway are linked to defective embryonic development

Prior to this, the following gaps must be filled to enable immune phenotype-guided clinical decision-making: (i) Using standardized testing protocols to longitudinally track the dynamic changes of these biomarkers, establishing their stable association with disease progression

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