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doi: 10.3390/nu6126020

In particular, the role of TLR4 is well known and in response to LPSs can trigger early and late activation of the pro-inflammatory NF-B transcription factor via TIRAP/MyD88 and TRAM/TRIF signaling cascades, respectively

The emphasis of this article was the therapeutic mechanisms of Rg1, which involved the reduction of inflammation, antioxidant properties, support for neuronal survival and regeneration, regulation of cellular energy processes, and enhancement of autophagic pathways

Cited 7/14/2021

By repressing the SLC7A11 gene at the transcriptional level, p53 contributes to the onset of ferroptosis by hindering the uptake of cysteine, an essential amino acid for GSH synthesis, thereby promoting ferroptosis (54)

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