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Hakulinen, J

In contrast, long-term cagrilintide treatment in rats upregulates prolactin-releasing hormone ( Prlh ) expression in nucleus of the solitary tract Calcr/Prlh cells that are conserved across rodents, macaques and humans

Lin et al., 2022) This oxidative dysregulation is thought to exacerbate neuroinflammation, mitochondrial dysfunction, and synaptic abnormalities, which may underlie some of the core behavioral and cognitive impairments seen in ASD (Gevezova et al., 2023)

doi: 10.3390/ijms21030836 81 GearingAJBeckettPChristodoulouMChurchillMClementsJDavidsonAHet al