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10.1002/ptr.7454 Phytother Res

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XPF silencing increases cisplatin induced cell death Since a higher amount of DNA damage, as shown by the H2AX analysis, correlated with increased cell death, we aimed to explore whether increased DNA repair capacity is responsible for A549 cisplatin resistance phenotype

This dissolution process also enhances the bioavailability of the peptide complex, allowing it to penetrate more effectively into the dermal layers where it exerts its regenerative effects

Meanwhile, studies have also identified a pathway that inhibits nuclear entry of NF-B through VPAC signaling, which inhibits IB phosphorylation (cAMP-independent pathway)