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(PubMed) Tanaka S, Haruma K, Yoshihara M, et al
PNAS Nexus 1, pgac248
DHT exerts anti-apoptotic effects and prevents H 2 O 2 - induced apoptotic cell death by activation of catalase, downregulation of p38 MAPK, JNK/SAPK and NF-kappaB via the androgen receptor
It is widely used in biochemical research as a selective, substrate-competitive inhibitor of nicotinamide N-methyltransferase (NNMT)
This can degrade the peptide, altering its structure and function
For long-duration GH axis studies, the long half-life reduces protocol variability
