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Diabetes increases expression of pro-apoptotic Bax and phosphorylation of JNK, while suppressing anti-apoptotic Bcl-2, thus promoting cell death of germ cells, Sertoli cells, and Leydig cells [241]
Aden D, Sureka N, Zaheer S, et al
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HO-1 is also involved in the protection of mitochondrial dysfunction and oxidative stress in diabetic Alzheimer's [165, 166]
