glutathione s transferase principle The role of glutathione-S-transferase in anti-cancer drug resistance Dual localization of glutathione S‐transferase

doi: 10.1016/j.brainresbull.2008.11.001

Late-stage oligodendrocyte progenitors are more sensitive to hypoxic or ischemic damage than early-stage progenitors and more mature oligodendrocytes [3]

The synergistic effect resulting from the depletion of GSH, regulation of acidity/metabolism, and the reinforcement of CT/CDT induced substantial mitochondrial damage and disturbed the redox homeostasis of cancer cells, ultimately exerting robust inhibitory effects on tumors

The rise in potassium depolarizes the cells to around -20mV (anoxic depolarization) and the release of glutamate [78]

881 Other HyT-based degraders are also developed, including those targeting HDAC and YEATS domain readers, providing therapeutic strategies for diseases caused by mutations or dysfunction in specific proteins

It is documented that neurons express G-CSF receptors, and exogenous G-CSF promotes peripheral nerve regeneration and function significantly [56]